phsl (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc manufactures this product 
96
Structured Review
Cell Signaling Technology Inc
phsl
Phsl, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 488 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+phsl/Phospho-HSL+(Ser563)+Antibody/bio_rxiv__64898__2026__03__20__712802-288-31-32
Average 96 stars, based on 488 article reviews
Phsl, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 488 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+phsl/Phospho-HSL+(Ser563)+Antibody/bio_rxiv__64898__2026__03__20__712802-288-31-32
Average 96 stars, based on 488 article reviews
phsl - by Bioz Stars,
2026-10
96/100 stars
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Related Articles
Membrane:Article Title: Sodium-potassium Adenosine Triphosphatase α2 Subunit (ATP1A2) Negatively Regulates UCP1-dependent and UCP1-independent Thermogenesis in 3T3-L1 Adipocytes Article Snippet: Increasing the number of brite cells (browning) in white adipocytes has attracted considerable attention to combat obesity because brite cells also help elevate energy expenditure.. Sodium-potassium adenosine triphosphatase α2 subunit (ATP1A2) has been studied extensively in migraine and cancers.. On the other hand, the role of ATP1A2 in adipocytes biology with a focus on fat browning needs to be elucidated. Article Title: Loss of family with sequence similarity 107, member A (FAM107A) induces browning in 3T3-L1 adipocytes. Article Snippet: Induction of white fat browning (beiging) and activation of brown fat has been considered a promising strategy to treat obesity and associated metabolic complications.. However, the molecular mechanisms regulating brown and beige fat-mediated thermogenesis remains unclear.. Our study aimed to identify genes with a hitherto unknown mechanism in the metabolic functions of adipocytes and identified family with sequence similarity 107, member A (FAM107A) as a factor that interferes with fat browning in white adipocytes. Article Title: ATP1A2 negatively regulates myogenic differentiation and thermogenesis in C2C12 myoblasts Article Snippet: It is well documented that the α2 subunit of Na+/K+-ATPase (ATP1A2) positively regulates obesity, cardiovascular disease, and diabetes, and additionally indirectly controls cellular Ca2+ homeostasis.. However, the functional role of ATP1A2 in the muscle remains unexplored.. Here, we investigated the functional role of ATP1A2 in muscle physiology with a focus on thermogenesis, using mouse-derived C2C12 myoblasts. Incubation:Article Title: Sodium-potassium Adenosine Triphosphatase α2 Subunit (ATP1A2) Negatively Regulates UCP1-dependent and UCP1-independent Thermogenesis in 3T3-L1 Adipocytes Article Snippet: Increasing the number of brite cells (browning) in white adipocytes has attracted considerable attention to combat obesity because brite cells also help elevate energy expenditure.. Sodium-potassium adenosine triphosphatase α2 subunit (ATP1A2) has been studied extensively in migraine and cancers.. On the other hand, the role of ATP1A2 in adipocytes biology with a focus on fat browning needs to be elucidated. Article Title: The development of metabolic endotoxemia is dependent on the type of sweetener and the presence of saturated fat in the diet. Article Snippet: .. Membranes were blocked in a blocking buffer consisting of 3% BSA in TBS Tween for 60 min at room temperature and incubated overnight at 4oC with the primary antibody and the following primary and secondary antibodies in the blocking buffer: Article Title: The development of metabolic endotoxemia is dependent on the type of sweetener and the presence of saturated fat in the diet Article Snippet: .. Membranes were blocked in a blocking buffer consisting of 3% BSA in TBS Tween for 60 min at room temperature and incubated overnight at 4oC with the primary antibody and the following primary and secondary antibodies in the blocking buffer: Article Title: Loss of family with sequence similarity 107, member A (FAM107A) induces browning in 3T3-L1 adipocytes. Article Snippet: Induction of white fat browning (beiging) and activation of brown fat has been considered a promising strategy to treat obesity and associated metabolic complications.. However, the molecular mechanisms regulating brown and beige fat-mediated thermogenesis remains unclear.. Our study aimed to identify genes with a hitherto unknown mechanism in the metabolic functions of adipocytes and identified family with sequence similarity 107, member A (FAM107A) as a factor that interferes with fat browning in white adipocytes. Article Title: A bioenergetic basis for multiorgan dysfunction in sepsis Article Snippet: .. Samples were incubated with anti-ATGL (PNPLA2/ATGL Antibody, Novus biologicals, Cat#: AF5365, 1:800) and Article Title: ATP1A2 negatively regulates myogenic differentiation and thermogenesis in C2C12 myoblasts Article Snippet: It is well documented that the α2 subunit of Na+/K+-ATPase (ATP1A2) positively regulates obesity, cardiovascular disease, and diabetes, and additionally indirectly controls cellular Ca2+ homeostasis.. However, the functional role of ATP1A2 in the muscle remains unexplored.. Here, we investigated the functional role of ATP1A2 in muscle physiology with a focus on thermogenesis, using mouse-derived C2C12 myoblasts. Blocking Assay:Article Title: The development of metabolic endotoxemia is dependent on the type of sweetener and the presence of saturated fat in the diet. Article Snippet: .. Membranes were blocked in a blocking buffer consisting of 3% BSA in TBS Tween for 60 min at room temperature and incubated overnight at 4oC with the primary antibody and the following primary and secondary antibodies in the blocking buffer: Article Title: The development of metabolic endotoxemia is dependent on the type of sweetener and the presence of saturated fat in the diet Article Snippet: .. Membranes were blocked in a blocking buffer consisting of 3% BSA in TBS Tween for 60 min at room temperature and incubated overnight at 4oC with the primary antibody and the following primary and secondary antibodies in the blocking buffer: Article Title: A bioenergetic basis for multiorgan dysfunction in sepsis Article Snippet: .. Samples were incubated with anti-ATGL (PNPLA2/ATGL Antibody, Novus biologicals, Cat#: AF5365, 1:800) and Cell-Signaling:Article Title: Semaphorin 7A regulates axon outgrowth in subcutaneous white adipose tissue Article Snippet: .. Primary antibodies: anti-Cyclophilin B (21 kDa, 1:40,000, Abcam, Cat#16045), anti-beta-tubulin (55 kDa, 1:1000, Cell Signaling, Cat#2146), anti-ATGL (55 kDa, 1:1000, Cell Signaling, Cat#2138), anti-HSL (81 kDa, 1:1000, Cell Signaling, Cat#4107), Saline:Article Title: ATP1A2 negatively regulates myogenic differentiation and thermogenesis in C2C12 myoblasts Article Snippet: It is well documented that the α2 subunit of Na+/K+-ATPase (ATP1A2) positively regulates obesity, cardiovascular disease, and diabetes, and additionally indirectly controls cellular Ca2+ homeostasis.. However, the functional role of ATP1A2 in the muscle remains unexplored.. Here, we investigated the functional role of ATP1A2 in muscle physiology with a focus on thermogenesis, using mouse-derived C2C12 myoblasts. |
